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Bodybuilders often take HGH in exogenous form to increase HGH production, increasing muscle mass and fat loss.[1] There is also little evidence comparing the effect of exogenous exogenous HGH and the use of LHRH and CORT in patients with PCOS.[4] It seems unlikely that elevated testosterone or estrogen in those with PCOS would lead to the development of insulin resistance and subsequent insulin resistance related to obesity. The majority of people with PCOS and insulin resistance could not benefit the development of insulin resistance from an elevated testosterone or estrogen levels. Many patients with PCOS do not experience menstrual cycles. Some also believe they experience irregular menstrual cycles, while others believe there are cycles that don't occur. However, those who experience the menstrual cycle syndrome with PCOS do not experience it in men and vice versa. In women of childbearing potential, there is a risk of ovarian hyperstimulation syndrome and ovarian hypoactive sexual desire disorder associated with ovarian hyperstimulation. This may be associated with an increased risk for ovarian cancer, although research has not been done to prove the link. There is one small trial that found increased odds for weight gain and insulin resistance in those with PCOS who took synthetic HGH.[5] This could be due to increased metabolic stimulation from the use of exogenous HGH or the use of higher doses of exogenous exogenous HGH. An alternative explanation could be that those with insulin resistance and obesity do not always experience an increase in insulin resistance during pregnancy in the same way that PCOS patients do. The use of exogenous HGH may interfere with hormone levels in areas of the body outside of the uterus and ovaries. Exogenous HGH may result in an increased production of IGF-1, a growth hormone that is used to promote growth of organs in body (such as fat-bearing organs). IGF-1 is not absorbed normally so in areas such as the liver and pancreas, IGF-1 is often elevated.[6] HGH also may have a negative effect to insulin sensitivity, as IGF-1 is known to inhibit insulin secretion. This has also been suggested by studies with animal models. For example, there is some evidence to suggest that exogenous exogenous HGH and/or insulin could affect insulin release.[7] Exogenous HGH's effect on insulin has not been studied in humans, but it is possible that HGH's effects might impact on the hypothalamic–pituitary–adrenal axis. Insulin resistance is a significant risk factor for PCOS. Insulin resistance is associated with obesity and Similar articles:

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